Does anyone have any clinical data/research/ideas as to why DVT"S develop within the first 2 weeks of a PICC insertion? I undestand high risk factors will contribute to this complication ie.. diagnosis on hosptial admission (ca, infection etc..) , catheter type - taper vs. non taper, power vs. non-power, bedrest, TPN infusion, insertion tech., flushing protocol, renal pts, multiple IV sticks etc... Are these hosptialized patients just at high risk? Any further insight is appriciated.